Asthma, accompanied by lung inflammation, bronchoconstriction and airway hyper-responsiveness, is a significant public health burden.  Currently, asthma treatments such as anticholinergics target the hypersensitive nerves which connect the brain to parasympathetic neurons in the smooth muscle of the airway.  Therefore, insights into the neural mechanisms underlying the pathogenesis of asthma are highly desirable.  Now, a study from researchers at Johns Hopkins