A team of scientists, led by researchers at The Wistar Institute, has found that an infection with herpes simplex virus 1 (HSV-1) causes rearrangements in telomeres, small stretches of DNA that serve as protective ends to chromosomes. The findings show that this manipulation of telomeres may explain how viruses like herpes are able to successfully replicate while also revealing more about the protective role that … Continue reading Researchers map previously unseen herpes-telomere interaction.