Neurodegeneration in Alzheimer’s disease is thought to be caused by the abnormal behavior of amyloid-beta molecules, which are known to gather into tough fibril-like structures called amyloid plaques within patients’ brains.  This widely held view has guided therapeutic strategies and drug development for more than 30 years.  Now, a study from researchers at Massachusetts General Hospital (MGH) provides additional evidence that amyloid-beta protein, which is … Continue reading Human amyloid-beta shown to act as a powerful antibiotic in the brains of animal models.