Alzheimer’s disease (AD) is characterized by cognitive decline, alteration of synaptic transmission, and neuronal death brought on by accumulation of neurofibrillary tangles in neurons and amyloid beta in the brain.  The leading hypothesis for the cause of AD is an impairment of clearance of amyloid beta resulting from the β-cleavage of the amyloid precursor protein. Recent studies showing that reducing amyloid beta load in mouse models improves … Continue reading Study shows that vesicles appear to also contribute to Alzheimer’s disease.